- Questo topic ha 7 risposte, 2 partecipanti ed è stato aggiornato l'ultima volta 1 giorno fa da RichardGeove.
18 Aprile 2014 alle 16:43 #23842Stefano CotterliOspite
This is not simply a request of a new feature: YOU ARE VIOLATING the TOU of the OpenStreetMap, missing the attribution. This problem was already pointed out by this post:
I am sure you plugin WAS displayng the correct attribution some versions ago, but now no longer. It is obviously a little bug you was not aware to introcuce, so fix it as up please!
Refers to: http://www.openstreetmap.org/copyright/en9 Giugno 2014 alle 12:13 #24158maxAmministratore del forum
Sorry, it’s not intention to break any license, I’ll fix asap9 Giugno 2014 alle 12:30 #24159maxAmministratore del forum
Wait wait, I’m not showing the credits and this is 100% my fault.
Are you sure I was displaying the correct attribution? I don’t like this kind of supposition.10 Agosto 2014 alle 12:00 #24284tcsindustryoOspite
Ciclosporin (INN/BAN) (pronounced /?sa?kl??sp?r?n/);[1 – cyclosporine (USAN); cyclosporin (quondam PROHIBITION); or ciclosporin A, cyclosporine A, or cyclosporin A (continually shortened to CsA) is an immunosuppressant cure largely reach-me-down in voice transplantation to intercept rejection. It reduces the endeavour of the exempt methodology during interfering with the activity and rise of T cells.[2 – [3 – It was initially isolated from the fungus Tolypocladium inflatum (Beauveria nivea), rest in a contaminate specimen obtained in 1969 from Hardangervidda, Norway, by Dr. Hans Peter Frey, a Sandoz biologist.[4 – Most peptides are synthesized by means of ribosomes, but ciclosporin is a cyclic nonribosomal peptide of 11 amino acids and contains a lone D-amino acid, which are rarely encountered in nature.[5 –
The immunosuppressive drift of ciclosporin was discovered on 31 January 1972 via employees of Sandoz (in the present circumstances Novartis) in Basel, Switzerland, in a screening test on immune stop designed and implemented not later than Hartmann F. St?helin, M.D. The happy result of ciclosporin in preventing structure rebuff was shown in kidney transplants past R.Y. Calne and colleagues at the University of Cambridge,[6 – and in liver transplants performed before Dr. Thomas Starzl at the University of Pittsburgh Hospital. The primary patient, on 9 Procession 1980, was a 28-year-old woman.[7 – Ciclosporin was later on approved as make use of in 1983. Since then, it has been utilized to forbid and investigate graft-versus-host reactions in bone-marrow transplantation and to ward rejection of kidney, centre, and liver transplants.[8 –
In wing as well as to transplants, ciclosporin is also used in psoriasis, severe atopic dermatitis, pyoderma gangrenosum, inveterate autoimmune urticaria, and, infrequently, in rheumatoid arthritis and related diseases, although it is on the other hand euphemistic pre-owned in wicked cases. It is commonly prescribed in the US as an ophthalmic emulsion for the treatment of plain eyes.[8 – It has been investigated on consume in sundry other autoimmune disorders, and is every once in a while prescribed in veterinary cases, uniquely in remotest cases of immune-mediated hemolytic anemia. Inhaled ciclosporin has been investigated to investigate asthma and is being contrived as a preventive therapy an eye to persistent brush-off of the lungs. Ciclosporin has also been reach-me-down to escape manage patients with acute uncompromising ulcerative colitis that do not respond to treatment with steroids.[9 – This psychedelic is also worn as a treatment of posterior or medial uveitis with noninfective etiology. Ciclosporin is a hallucinogenic currently acclimatized to experimentally handle cardiac hypertrophy[10 – [11 – (an growing in stall volume).
Ciclosporin has been investigated as a possible neuroprotective delegate in conditions such as wounding perspicacity offence, and has been shown in animal experiments to modify brain reparation associated with injury.[12 – Ciclosporin blocks the array of the mitochondrial permeability change pore, which has been found to issue much of the damage associated with chief executive officer maltreatment and neurodegenerative diseases. Ciclosporin’s neuroprotective properties were first discovered in the inappropriate 1990s when two researchers (Eskil Elmér and Hiroyuki Uchino) were conducting experiments in cubicle transplantation. An unintended finding was that CsA was strongly neuroprotective when it crossed the blood–brain barrier.[13 – This unvaried answer of mitochondrial breaking up fully the rent of the MPT pore is implicated in making harmful intellect injuries much worse.[14 – Cyclosporine is currently in a appearance II/III (adaptive) clinical study in Europe to adjudge its know-how to ameliorate neuronal cellular damage in traumatizing genius injury. This multi-center about is being organized by means of NeuroVive Pharma and the European Brain Mistreatment Consortium using NeuroVive’s formulation of cyclosporine called NeuroSTAT?. [15 – From http://www.tcsindustry.com – TCS INDUSTRY LTD23 Agosto 2014 alle 03:26 #24331fmyykjbOspite
Daptomycin is a cyclic lipopeptide antibiotic produced by the organism Streptomyces roseosporus.[14 – [15 – Daptomycin consists of thirteen amino acids, ten of which are arranged in a cyclic fashion, and three that adorn an exocyclic tail. Two non-proteinogenic amino acids exist in the lipopeptide, the unusual amino acid L-kynurenine (Kyn), only known to Daptomycin, and L-3-methylglutamic acid (mGlu). The N-terminus of the exocyclic tryptophan residue is coupled to decanoic acid, a medium chain (C10) fatty acid. Biosynthesis is initiated by the coupling of decanoic acid to the N-terminal tryptophan, followed by the coupling of the remaining amino acids by nonribosomal peptide synthetase (NRPS) mechanisms. Finally, a cyclization event occurs, which is catalyzed by a thioesterase enzyme, and subsequent release of the lipopeptide is granted.
The non-ribosomal peptide synthetase (NRPS) responsible for the synthesis of Daptomycin is encoded by three overlapping genes, dptA, dptBC and dptD. The dptE and dptF genes, immediately upstream of dptA, are likely to be involved in the initiation of daptomycin biosynthesis by coupling decanoic acid to the N-terminal Trp.[16 – These novel genes (dptE, dptF ) correspond to products that most likely work in conjunction with a unique condensation domain to acylate the first amino acid (tryptophan). These and other novel genes (dptI, dptJ) are believed to be involved in supplying the non-proteinogenic amino acids L-3-methylglutamic acid and Kyn; they are located next to the NRPS genes.[16 –
From http://www.fmyykj.com – suzhou pharmaceutical technology co.,ltd.12 Settembre 2014 alle 09:43 #24345famuyiyaouOspite
Moxidectin (Milbemycin B[1 – ) kills parasites, so is used because of the arrest and repress of heartworm and intestinal worms, and can be found in treatments prescribed to go to animals such as dogs, cats, horses, cattle and sheep. Relevancy methods towards moxidectin change before treatment, and contain uttered, current, and injectable solutions. Cydectin Pour On is a swap cite towards a formulation fitted use on cows and red deer.
Moxidectin is a semisynthetic imitative of nemadectin [2 – which is produced during fermentation at near Streptomyces cyano-griseus. This Streptomyces species was discovered in a soil sampler from Australia in the fresh 1980s unperturbed by means of an agronomist working for the American Cyanamid company.
Moxidectin treats and controls some of the most mean internal and outward parasites by way of selectively binding to parasites’ glutamate-gated chloride ion channels. These channels are mandatory to the go of invertebrate balls and muscle cells; when moxidectin binds to the channels, it disrupts neurotransmission, resulting in paralysis and cessation of the parasite.
Studies of moxidectin posture the side effects restyle sooner than animalistic and may be fake by the product’s formulation, pertinence method and dosage. The products are usually recommended close a veterinarian to make sure correct avail oneself of and application. Herding dogs may be avermectin-sensitive, but avermectin-sensitive dogs can admit standard doses after heartworm prevention. Moxidectin is manifestly unharmed in regard to collie breeds. As a heartworm preventative, moxidectin can be injected once every six months at the beck the sort big cheese Proheart6, or every 12 months answerable to the brand eminence Proheart SR 12.
Moxidectin is the subject of a trial to assess its suitability, as an alternate to ivermectin, to treat onchocerciasis in humans.[3 –
From http://www.fmyykj.com – suzhou pharmaceutical technology co.,ltd.23 Settembre 2014 alle 14:26 #24359tcsgrouphOspite
yes, i’m agree with you!
From http://www.tcsindustry.com – TCS INDUSTRY LTD18 Aprile 2019 alle 08:23 #84873FrankHopOspite
Can you plese welcome me, i’m a new user